- Reaction score
- 2,035
I have been telling everyone on here that AA's are not necessary since I signed on. AA's for the most part only exist to reduce the amounts of estradiol needed to reach levels. The reason that one used to especially limit estradiol/estrogens is because the main two medications with an estradiol-like structure, were one synthetic, in ethinyl estradiol or two, contained substantial non-human equine estrogens. These were much less safe in terms of blood clots and liver damage.So you are saying that E2 alone is enough to regrow hair to a satisfactory state? I know about the trans people using E2 only, even some online pharmacies state that monotherapy is the best way for feminization. But I'm not trans, I'm looking at this from a hair angle. And after all this disease is called Androgenic Alopecia, and a lot of scientific evidence points to the fact that all Androgens, not only DHT but also T play a role in hair loss, especially in people with over sensitive genes, like me. So going with that line of thought I decided that I want to rid my body of all Androgens as much as I can for at least 9-12 months. But I won't lie, what you are saying is intriguing, I think you are the first person here saying that they use only Estradiol without anything else. There are people who don't use AAs, but they still use 5-ARIs from what I gathered going through this thread.
Those two meds continue most likely to be the highest sold HRT/birth control meds in the world but virtually everyone with means or access uses bio-identical estradiol. AA's appear to do very little for hair growth sans estrogen and what they do do, is probably by raising estrogen itself. To his credit, Almas went round and round on this issue with me and with a couple of other folks that I don't see on here as much anymore, one of them an intern to be a physician in Brazil. You can grow hair fine with AA's but no one has articulated any process by which they might be necessary save for those who don't respond to estrogen. AA's might get a person to limits quicker but patches and injections and Estrogel probably do this as quickly as anyone might want.
I do use dutasteride/finasteride but that is for DHT, not testosterone lowering. Duta/finasteride both seem to be inclined to raise T some. DHT also to a certain extent provides a floor should things not proceed in terms of hair loss. There's zero reason to believe in my estimation that low-dose estradiol is better for hair results than is high dose. Low-dow is indicated for those seeking the best breast growth possible. High-dose seems to feminize more quickly for facial feminization and hair. But most XY's probably don't want either facial feminization or breasts and Bridge got both, large amounts of both, and he used large amounts of AA's.
Most people on HRT believe that AA's are tools, somehow, arrows in our quiver to chase or modify results. They are not and adding an extra med might not hurt but again it might but you actually know less about what is going on with the more implicated meds. Anyone thinking about only AA's, as was even recent thought on here six months back, should desist. It won't work to regrow hair and it might led to a massive shed. To do this, optimally, you need one "male" med, dutasteride, one female med, estradiol, one hair growth med, minoxidil and everyone should microneedle once weekly. If there are signs of inflammation and for shed control, add keto.
That's far more simplified than trying to manage AA and estradiol dose. Do those four things/steps and it either works or it doesn't. There appears to be no other way, save Serms, which present even more meds that are uncertain in effect to deal with.
Last edited: