Bryan said:
DarkVctry said:
The thing that tricks people is.....it has GLA's and they are proven to be extremly effective in combating DHT. However it must be FREE FORM GLA's. Revivogens GLA's/LA's are not.
Revivogen uses free form fatty acids. Where did you get the impression that they don't? :dunno:
Scalp skin can make the hidrólisis of triglycerides:
The role of bacteria in the formation of free fatty acids on the human skin surface
1. Human skin surface and hair can be rather
easily sterilized by local application of antibiotic
preparations.
2. The lipolytic activity of the skin surface is
diminished after sterilization of the skin.
3. It is concluded that bacteria participate
in the production of free fatty acids on the skin
surface.
4. In addition to bacterial activity, the presence
of lipolytic esterases of cellular origin is
assumed because by sterilization of the surface
its lipolytic activity is never completely abolished
Other study:
http://www.nature.com/jid/journal/v62/n ... 7560a.html
Genesis of free fatty acids
According In our current state of knowledge, free fatty acids are formed in sebaceous follicles primarily through hydrolysis of sebum triglycerides by microbial lipases. Because of their comedogenic and irritant qualities, these free fatty acids are probably responsible for many of the alterations in the follicle that result in acne lesions.
Others references:
• Marples, RH, Downing, DT, Kligman, AM: Control of free fatty acids in human surface lipids by Corynebacterium acnes. J. Invest. Dermatol 1971 56: 127–131,
• Marples, RR, Downing, DT, Kligman, AM: Influence of pityrosporum species in the generation of free fatty acids in human surface lipids. J. Invest. Dermatol 1972 58: 155–159,
• Marples, RR, Kligman, AM: Ecological effects of oral antibiotics on the microflora of human skin. Arch. Dermatol 1971 103: 148–153,
• Marples, RR, Kligman, AM, Lantis, LR, Downing, DT: The role of the aerobic microflora in the genesis of fatty acids in the human surface lipids. J. Invest. Dermatol 1970 65: 173–177,