Valporic acid (Feb 2014, in vivo)

waynakyo

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[h=1][FONT=Verdana, Arial, Tahoma, Calibri, Geneva, sans-serif]opical valproic acid increases the hair count in male patients with androgenetic alopecia: a randomized, comparative, clinical feasibility study using phototrichogram analysis.[/FONT][/h][FONT=Verdana, Arial, Tahoma, Calibri, Geneva, sans-serif]Jo SJ1, Shin H, Park YW, Paik SH, Park WS, Jeong YS, Shin HJ, Kwon O.[/FONT]
[h=3][FONT=Verdana, Arial, Tahoma, Calibri, Geneva, sans-serif]Author information[/FONT][/h]

[h=3][FONT=Verdana, Arial, Tahoma, Calibri, Geneva, sans-serif]Abstract[/FONT][/h][FONT=Verdana, Arial, Tahoma, Calibri, Geneva, sans-serif]Valproic acid (VPA), a widely used anticonvulsant, inhibits glycogen synthase kinase 3β and activates the Wnt/β-catenin pathway, which is associated with hair growth cycle and anagen induction. To assess the efficacy of topical VPA for treatingandrogenetic alopecia (Androgenetic Alopecia), we performed a randomized, double-blind, placebo-controlled clinical trial. Male patients with moderate Androgenetic Alopecia underwent treatment with either VPA (sodium valproate, 8.3%) or placebo spray for 24 weeks. The primary end-point for efficacy was the change in hair count during treatment, which was assessed by phototrichogram analysis. Of the 40 patients enrolled in the study, 27 (n = 15, VPA group; n = 12, placebo group) completed the entire protocol with good compliance. No statistical differences in age, hair loss duration and total hair count at baseline were found between the groups. The mean change in total hair count was significantly higher in the VPA group than in the placebo group (P = 0.047). Both groups experienced mostly mild and self-limited adverse events, but their differences in prevalence rates were similar between the two groups (P = 0.72). A subject treated with topical VPA developed ventricular tachycardia, but it did not seem to be related to the VPA spray. Topical VPA increased the total hair counts of our patients; therefore, it is a potential treatment option for Androgenetic Alopecia.[/FONT]

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Was this posted before? it seems now there is a string of papers showing efficacy of VPA although this is the first in male patients I think (?)

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Oh if anyone has access to the full text pls share, wonder whether these are terminal hairs...
 

RK85

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Some interesting post on BTT forum:

boldy said:
thanks for posting! I've read the study, however this isn't really new news, if we have to translate this info into human-model or chemically induced wnt activation which is shown previously.

the fact that expression or over expression of wnt-beta cetanin in some compartments of the hairfoliclem can induce anagen phase is showen in other studies, that used GSK3b inhibitors, which can overrule Dkk1 inhibitory signals, since they don't have to trigger Frizzled, and lrp 5/6 receptors.

there are also gsk3b inhibitors, which you can get ovr the counter, like Lithium, which can do the same, inducing lef-1 and ALP, in vivo and vitro.


other powerful inhibitor is FDA approved compound is: Sodium valproate /valproic acid (VPA). there is a human trial conducted, which showed positive effect which show some positive effect in humans.

https://www.dropbox.com/s/klnqvyzqw...oic acid increases the hair count in male.pdf

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When is valproic acid taken orally, it can have serious side effects:

http://en.wikipedia.org/wiki/Valproic_acid#Adverse_effects

Does the topical use of valproic acid also have these side effects?
 
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