Exploring The Hormonal Route. Hair=life.

JaneyElizabeth

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Yeah but the point is that even more so, they are going to avoid bica since it raises E and blocks receptors for AR.
We should just make all XY's go on MtF HRT and then, later, if they want to breed, they could go off HRT temporarily to inseminate and then back on HRT to protect hair. It's a life-time program....
 

Pls_NW-1

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We should just make all XY's go on MtF HRT and then, later, if they want to breed, they could go off HRT temporarily to inseminate and then back on HRT to protect hair. It's a life-time program....
After 8-12 months testes will have irreversible damage. Won't be that easy I guess haha
 

2TameDHT

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I have been equating 1 drop with 1 gram of loniten but drops probably aren't an efficient dosing mechanism. The problem is that most droppers only mark down to 25 ml which is equated to 12.5 mg of Loniten.

Maybe you can explain the paragraph below because I have never heard a good explanation of why some doctors start off HRT with only an AA for sixty to 90 days instead of just going with estradiol monotherapy.

I am thinking that when I start, I may want to do an AA for the first three months to give the estradiol an edge over DHT and T before continuing with estradiol monotherapy, as well as continue use of Finasteride or get Dutasteride to better nuke DHT with. I'll incorporate minoxidil solution ingestion pretty soon. From what I've read on this thread one drop of liquid minoxidil is equivalent to about 1.25 mg oral minoxidil.
From what I can tell, it may be for those with particularly high Testosterone. And/or those who want the estradiol to have less opposition so that it may be more effective early on. A lot of it may simply be personal choice.

I would get Loniten/generic oral minoxidil, but that looks to be more difficult to get a hold of.
 

JaneyElizabeth

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From what I can tell, it may be for those with particularly high Testosterone. And/or those who want the estradiol to have less opposition so that it may be more effective early on. A lot of it may simply be personal choice.

I would get Loniten/generic oral minoxidil, but that looks to be more difficult to get a hold of.
Drizzling the topical on the back of the tongue even two drops daily is essentially free if someone has topical min anyway but regardless, these bottles are like six bucks a piece and last forever when used to dose mere drops. It's hard to believe just how little oral (topical) min is needed to see results, even substantial ones.
 

DogoDiLaurentiis

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This reinforces what I think, the treatment has to be exclusive to you, it will be different for each person, you can't say and offer treatment to anyone and you can't trust it either, I don't know any of the 2 or who posted or DHTcel is one of our examples of results, and notice our few results are surrounded by doubts and disagreements haha

As I said I don’t support any speech I just posted because I hadn’t seen his photos DHTcell Early too and I found the comment of the user who posted relevant, with a lot of links and blah blah

Peace my warriors

Absolutely, in the end I think in part my first recovery was 50% luck and 50% having a very general understanding of what was causing it.

This second incidence has compelled me to do a lot more research and experimentation, what works for myself may or may not work for another, what I do however to help others is give context.

So when I add a whole bunch of other health or biology related anecdotes, it's to help people understand a bit more about the reasons why something may or may not work for me particularly, if they fit the model of what I present, it may help them too.

In the end, because medical research and healthcare response to hair loss is marginal to say the least, all of us in our own way have to be researchers of both the things we're going to take, and how our bodies work individually. We share information because like all other living things on this earth we are pattern based beings, and some aspects of our experience and biology may overlap.
 

Almas

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Apparently, I avoided gynecomastia. It always starts to show in the first month, but I have no signs. Good or bad - I don't know. If it doesn't stop baldness, I'll switch to 75mg.
According to my small statistics, the T level at the age of 16-20 years when taking Bicalutamide rises to 1200ng / dl
 

Gergely

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Apparently, I avoided gynecomastia. It always starts to show in the first month, but I have no signs. Good or bad - I don't know. If it doesn't stop baldness, I'll switch to 75mg.
According to my small statistics, the T level at the age of 16-20 years when taking Bicalutamide rises to 1200ng / dl
hmm i didn't had gyno for the first 2 months i believe, hard to remember.
 

Almas

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So you think that the 62 percent on my thread that indicate "dude, I won't even touch finasteride" are going to use bicalutamide instead when that is actually a hormonal medication that can cause feminization?
If a person is not ready to sacrifice something for the sake of hair, then this is a small problem
 

Pls_NW-1

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Apparently, I avoided gynecomastia. It always starts to show in the first month, but I have no signs. Good or bad - I don't know. If it doesn't stop baldness, I'll switch to 75mg.
According to my small statistics, the T level at the age of 16-20 years when taking Bicalutamide rises to 1200ng / dl
Crazy... I wonder how you avoided gyno!? And damn that T level is very high.
 

Almas

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Crazy... I wonder how you avoided gyno!? And damn that T level is very high.
This is not my T. This is the average T in young people after taking Bica. I don’t know my T, but I’m sure it is also near this value.
According to statistics, gynecology at 50mg in 36% of people. No wonder I don't have it
 

Almas

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I have known someone who has been using 50mg Bica + 2 pressure of Estrogel for two months now. He also does not have gynecomastia.
 

Pls_NW-1

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People are saying to me, I shouldn't use Finasteride... while I have friends who are 16 years old taking Dutasteride... :eek:.

I should remind that maintenance is the key, significant cosmetic regrowth is very hard...
 

Almas

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The percentage of gynecomastia does not increase at dosages above 100 mg. Apparently, there is no point in exceeding this dose. Perhaps I do not have gynecomastia, because 50mg cannot block all androgens, and they continue to displace E in the breast tissue. Let's see what will happen next. I will increase the dosage to 75mg in the fall if I still don't see a change.
 

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Pls_NW-1

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The percentage of gynecomastia does not increase at dosages above 100 mg. Apparently, there is no point in exceeding this dose. Perhaps I do not have gynecomastia, because 50mg cannot block all androgens, and they continue to displace E in the breast tissue. Let's see what will happen next. I will increase the dosage to 75mg in the fall if I still don't see a change.
I wonder what for an effect 75mg has... 100mg seems like the ideal dose as a monotherapy.

However, we have no datas for 75mg.
 
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