Exploring The Hormonal Route. Hair=life.

bridgeburn

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Hello Brige!
Thanks for answer, but I think it will be depend on dosage, maybe 2 mg of estrogen will not dangerous.
Probably not, but E is stronger when combined with an AA
 

Yar

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I used Flutamide for 6-8 years and it works like magic.
your estradiol production is important here, if your testicles produce estradiol then it will work for you. if you have FSH it will work androgen aromatization
 

KSA

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I'm 21 yo and I was on 50 mg CPA for about 2,5 months and I'm still losing hair.
My chest hair are almost gone, my skin is the best in my entire life, dick works good, i've become more emotional and I'm still losing ground.

I've been on check-up and my T is 14 (8-30 is normal) and liver is all good.

It's hard to say for me but probably nothing will save me. I'm gradually losing all hope and motivation.

I'm going to increase CPA dosage to 75 -> then check-up -> then add estradiol, but I don't believe that ANYTHING will save my hair anymore.
Honey no! That is not nearly enough time. Just be consistent and wait.
 

KSA

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your estradiol production is important here, if your testicles produce estradiol then it will work for you. if you have FSH it will work androgen aromatization
Not really. Flutamide is a STRONG anti androgen at the right dose for your body. It is oral RU or bicalutamide. My e was a 30pg/ml the entire period I took Flutamide. My T was well off the charts as was my DHT.
 

KSA

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I've always thought the science behind bica seems sound. But I'm hard pressed to find very many success stories and it seems woefully underused if it's superior to every other method. Why do you think that is?
It is not regularly used in the dermatological environment. In fact, both Flutamide and bicalutamide are very limited even in the gynaecological environment at this moment in time. Perhaps a clever pharma company (heyyyy Purdue) may swoop in to exploit
 

Yar

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Not really. Flutamide is a STRONG anti androgen at the right dose for your body. It is oral RU or bicalutamide. My e was a 30pg/ml the entire period I took Flutamide. My T was well off the charts as was my DHT.
flutamide is not used hrt mtf, it is not prescribed mtf endocrinologists
 

Yar

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Not really. Flutamide is a STRONG anti androgen at the right dose for your body. It is oral RU or bicalutamide. My e was a 30pg/ml the entire period I took Flutamide. My T was well off the charts as was my DHT.
flutamide does not use HRT mtf, it is not prescribed by mtf endocrinologists
 

Father_of_Shiseido

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It is not regularly used in the dermatological environment. In fact, both Flutamide and bicalutamide are very limited even in the gynaecological environment at this moment in time. Perhaps a clever pharma company (heyyyy Purdue) may swoop in to exploit
How much flutamide feminized you?
 

KSA

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flutamide does not use HRT mtf, it is not prescribed by mtf endocrinologists
Yes because it is non steroidal - so while it can feminize you, you can’t track your levels. In fact, my idiot doctor who rxed ne thought I had CAH when I started taking Flut.
How much flutamide feminized you?
500mg is the standard dose. Maybe too much. I couldn’t tolerate it so I would drop to 250 - then experience a massive shed / breakout and titrate up. It’s superior to spironolactone for sure, but with that said spironolactone won’t elevate your androgens thiiiiis much. Maybe flut with some e could help that reflex but it’s otherwise given to cancer patients who have taken GNRH analogs
 

Yar

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Yes because it is non steroidal - so while it can feminize you, you can’t track your levels. In fact, my idiot doctor who rxed ne thought I had CAH when I started taking Flut.

500mg is the standard dose. Maybe too much. I couldn’t tolerate it so I would drop to 250 - then experience a massive shed / breakout and titrate up. It’s superior to spironolactone for sure, but with that said spironolactone won’t elevate your androgens thiiiiis much. Maybe flut with some e could help that reflex but it’s otherwise given to cancer patients who have taken GNRH analogs
spironolactone is not a steroid prescribed by endocrinologists,
 

Derelict

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So i found a study with a guy who was using spironolactone for a year, developed gyno and it went away three months after stopping it, i plan on losing a lot of weight in that sorta time period so that works out alright. I will reconsider adding spironolactone back to my regimen then.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5846220/
 
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Michael1986

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I can write you any research in order to launch the drug on the market! Show your results before and after
The fact that finasteride and dutasteride are effective treatments for male pattern baldness is a very well established fact backed up by a large amount of scientific and anecdotal evidence. If these drugs didn't work, it is fairly obvious that we would all know this by now. Newcomers reading through all these threads need to be made aware of the fact that these drugs do work.
 

Derelict

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The fact that finasteride and dutasteride are effective treatments for male pattern baldness is a very well established fact backed up by a large amount of scientific and anecdotal evidence. If these drugs didn't work, it is fairly obvious that we would all know this by now. Newcomers reading through all these threads need to be made aware of the fact that these drugs do work.

Yes finasteride/dutasteride along with perhaps minoxidil should always be the first line of defence, but it's very rare to regrow temples etc the way bridge did with those methods.
 

Michael1986

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Yes finasteride/dutasteride along with perhaps minoxidil should always be the first line of defence, but it's very rare to regrow temples etc the way bridge did with those methods.
Yes I agree that temple regrowth is difficult with 5ar inhibitors. Crown regrowth is a little easier. I was fortunate enough to get some temple regrowth on dutasteride, but I know that this appears not to be the norm. I'm sure that HRT treatments probably are more effective than finasteride/dutasteride, but for most hair loss sufferers (myself included), they would not be viable treatment options due to their side-effect profiles.
 
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Father_of_Shiseido

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Yes because it is non steroidal - so while it can feminize you, you can’t track your levels. In fact, my idiot doctor who rxed ne thought I had CAH when I started taking Flut.

500mg is the standard dose. Maybe too much. I couldn’t tolerate it so I would drop to 250 - then experience a massive shed / breakout and titrate up. It’s superior to spironolactone for sure, but with that said spironolactone won’t elevate your androgens thiiiiis much. Maybe flut with some e could help that reflex but it’s otherwise given to cancer patients who have taken GNRH analogs
I asked how much flut feminised you while you were on it.
 

ali.talebi1994

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Yes I agree that temple regrowth is difficult with 5ar inhibitors. Crown regrowth is a little easier. I was fortunate enough to get some temple regrowth on dutasteride, but I know that this appears not to be the norm. I'm sure that HRT treatments probably are more effective than finasteride/dutasteride, but for most hair loss sufferers (myself included), they would not be viable treatments options due to their side-effect profiles.
temple regrowth is more likely to happen with the help of microneedling
 

Ikarus

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The fact that finasteride and dutasteride are effective treatments for male pattern baldness is a very well established fact backed up by a large amount of scientific and anecdotal evidence. If these drugs didn't work, it is fairly obvious that we would all know this by now. Newcomers reading through all these threads need to be made aware of the fact that these drugs do work.

Actually, I believe 5AR inhibitors should be classed as maintenance drugs considering that dominates the statistics. Combining a maintenance drug along with something which helps with regrowth (minoxidil) is what leads to most of the success stories surrounding 5AR inhibitors on this website.
 

Derelict

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Actually, I believe 5AR inhibitors should be classed as maintenance drugs considering that dominates the statistics. Combining a maintenance drug along with something which helps with regrowth (minoxidil) is what leads to most of the success stories surrounding 5AR inhibitors on this website.

Yeah for a good chunk of people they only really maintain, or the hair count increase is so small it doesn't make a cosmetic difference. I think if you are not afraid of sides and value your hair more aggressive drugs are useful to know and discuss about in fighting hair loss, but i do agree with michael and disagree with yar that finasteride and dutasteride are useless, they should always be the first option people go to. Like i said i just stopped spironolactone because i have pretty severe gyno(although it may look worse due to my weight) im willing to tolerate mild to moderate gyno whereas someone else might not be. It all comes down to the sides and how much you are willing to put up with to regain your hair and taking the appropriate steps to fight this horrible condition.
 
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